las

Welcome all seeking refuge from low carb dogma!

“To kill an error is as good a service as, and sometimes even better than, the establishing of a new truth or fact”
~ Charles Darwin (it's evolutionary baybeee!)

Monday, October 19, 2020

Jimmy Moore -- Big Fat Liar -- Steps in it AGAIN! It Was NOT a Joke, He Is!

Hey all!  Long (LONG) time no blog.      This is mostly an update, because the King Kahuna of Keto is still sort of kicking around out there.   

Jimmy Moore can't help himself!!



Nah ... not one iota of evidence!  LOLOL



On the gossip front, it appears the split with his wife is imminent and he lost the Low Carb Cruise and most of the friends in the split.  Christine has lost 400 lbs in the past year -- well, 50 of her own since no longer eating like Jimmy ... and Jimmy.  

After taking six or four months off, King Krankypants of Ketonia tried to rebrand as "The Man of Real" discussing his childhood.   That went over like a lead balloon and apparently what remaining sponsors he had went the way of handshakes and hugs in 2020.  

This has left Jimmy in three-to-four tee shirts, surviving on eggs from his chickens (and the occasional ribeye from a friend), ranting away on IG and solidifying his place in nutritional history proving that it is indeed cortisol and not calories or even insulin that causes fat to accumulate on his body.

Ever the victim, there are still a few people who believe his lies. His latest:
https://www.instagram.com/p/CGZ6e0EgfKr/?igshid=167iixnqqd0pb

Several years ago I posted a picture of myself with a stick of butter on my plate while making a funny face as a joke and that photo very quickly (and infamously) became an instant meme where people believed I was supporting eating that way at every meal. Seven years later, I still get asked about this! When people ask me why I promote guzzling butter, I simply respond that I don’t. Unfortunately, that’s not good enough. ⁣

One of the great lessons I have learned being in the nutritional health community for as long as I have is that people will always attempt to smear and characterize you in the way they want others to perceive you. It really doesn’t matter at all if what they are saying is true or not. The only objective is to fully discredit you so as to diminish the impact of your work. This butter guzzling lie is merely one example of this horrible practice that I’ve had to deal with. ⁣

We see this devious tactic played out often throughout life. When someone starts accusing someone else of something completely absurd and then continues to ask them about it time and time again even though it has been thoroughly and repeatedly denounced, the primary purpose is nothing more than ridicule, embarrassment, and personal destruction. The reasons for this can run the gamut, but I’ve seen it happening way too often. ⁣

When you’re the one being accused of something that’s simply not true, it’s almost impossible not to get at least a little bit defensive. Especially when you feel like you have repeatedly responded to the exact same inquiry as if you’ve never said anything at all—it wears on you! If you remain silent about it when people ask, then they’ll just assume their charge is true. So you have to keep repeating your answer. It’s a vicious cycle. ⁣

In my case with the butter guzzling charge, anyone who regularly follows my work closely will see absolutely zero evidence that I promote this insane practice. But that doesn’t stop people who would prefer that I shut up about everything. And in the end, that really is the only objective of repeating a lie that has been thoroughly debunked again and again. Question asked, question answered—NEXT!

So enjoy the flashback, or perhaps it's your first time seeing this.   






Original post:  April 2018


Blogstress Note:



A quick note to all:  Please share!!

An apology:  Parts are long, and I threw a bunch of images in at the end and such just to preserve for posterity.  Sorry!


An open letter to my critics who may be in the audience:

It's OK for you to think what you may of me.  If you find yourself wanting to blame me, realize this is exactly what Jimmy Moore wants from you so that he can continue perpetrating his fraudulent livelihood.

Jimmy Moore, and now his wife Christine Moore, are not really interested or seeking optimal health.  Jimmy has long given up on weight loss, and it matters not what the actual weight is on the scale at this point, he's behind the ball position on January 1, 2004 (when he began Atkins) in so many ways.  He has a serious eating disorder at the root of all of this ... something he shows no desire to address.  Very sad for him and those who love him. 

Still, he inexplicably preys on desperate obese people seeking solutions or excuses.  He does this to feed an insatiable need for accolades from others, and to live a higher life than he otherwise would be (like multiple cruises each year!).

He makes carbohydrate restriction-based plans -- I don't care what the next fad (and that's what it is!) incarnation is -- look bad.

If keto or LCHF or Atkins or whatever has worked for you, and continues to work for you, go for it!  I'm NOT anti-LC.  But do not spread junk science.  Do not follow and elevate the likes of Jimmy Moore who is FAILING eating that way.   Go forth and share your own story if you want.  Or not.  Probably not a good idea to turn it into your primary source of income.  If you disagree with my assessment of "failing", step back, imagine if he's any fill-in-the-blank spokesperson for some diet you think is bull$#!t and see if you wouldn't agree.    He is the one delegitimizing your successful way of life in the eyes of others.  He is the one adding to all the confusion by spouting nonsense like steak is chocolate cake (true story).

Carbohydrate restriction plans could gain more mainstream traction if its promoters, ESPECIALLY doctors and researchers, stuck to the facts and what the science shows.  It's just that won't make those folks rich, and the accepted science will be nowhere near the exaggerated claims you've heard repeated in your echo chambers. 

Lastly, if you know someone who follows Jimmy or is thinking of following him, share this post with them.  If they still feel he's a reliable source of information, at least you've done your part.  I'd rather you encourage them to chase success.



I don't know what exactly prompted him to try to claim his butter eating stunt was a joke, but that lie don't fly!  In the past with Kimkins and the David Duke KKK scandals, he had to address controversy.  But this must be coming in reaction to "friendly fire" -- criticism from within the community.  I am encouraged by this development!

It was no joke, Jimmy Moore is the joke

That (other) time when Christine Moore watched Jimmy Moore eat an entire stick of Kerrygold at his momma's house


Tuesday, January 21, 2020

What's the Problem with Nutrition Research? Is it True Health Iniative or NutriRECS?


Hello World .. and anyone still reading this blog!!

Yes, it's been forever.  I've been generally pretty well.  Knocking about on social media (Facebook and some Twitter) ... occasionally getting the urge to blog, write something, get to 80% then lose the mojo.   What has turned into a long hiatus wasn't planned, but it happened due to running out of steam and still seeing the same old, same old.  I may or may not discuss more at some point, but if I don't just jump back in and blog, I probably never will again!

And so, with that said ...

𐩘𐩘𐩘𐩘𐩘𐩘𐩘𐩘𐩘𐩘𐩘


The following editorial in JAMA has been making the rounds on my Facebook feed, and I have a bit of a different take and reaction to it ... one that comes from roughly a decade now of delving into dietary recommendations wars from where I "stand".



I would encourage everyone to read this before this blog post.  It's interesting to me how differently people react and would love if you join in comments.


Before I begin here, this post will not be discussing red meat studies and the healthfulness per se.    I also want to make it clear that this is not an endorsement of Dr. David Katz or his True Health Initiative.   But it is also crystal clear where Katz and the THI's biases lie ... the message is not hidden on their website:  True Health Initiative.


This is the current Top Story on the site.


Tuesday, April 2, 2019

Why We Get Fat ... Lessons from a Cafeteria Rat

BUMP!

In addition to blogging on several topics raised during Stephan Guyenet and Gary Taubes recent appearance on the Joe Rogan Experience podcast, I've decided to bump some of the past content on this blog relevant to some topics discussed and/or research studies mentioned.  I'll include a little commentary with each.

At around the 12 minute mark, Stephan makes a great opening play, and then gets distracted or flustered into fumbling the football.  Stephan asks what it is about the food environment, etc. that causes obesity, and distills it down to the simplest of questions (paraphrase):
Q:  What is the most fattening diet on the planet?
A:  Human junk food.
Nail.  Head.  Boom.  He then calls out some numbers from the research summary he posted on his blog in advance of the podcast.   The study I discuss in this blog post is the one included in "#52" that he's discussing in the podcast.    Joe seemed perplexed by this idea that we couldn't replicate the junk food fattening with sugar or even fat alone.   Joe seems fixated on junk food = sugary "bullshit diet".  He gets very confused, despite Stephan laying it out fairly well.  Give just sugar/refined carb or give just fat, and the rats don't get AS obese as they do when you give them a mixture of sugar/fat and FLAVOR in human junk foods.    Yes, Joe, soda is just sugar and is a junk food, but junk foods are  also things like donuts, cookies and ice cream or pizza, fries and Doritos.  Carb + fat + flavor. 

While the CAF rats are indeed rats, this study does a fairly good job in demonstrating that it's not just the macros.     Four diets:  Standard Chow (mostly grains, 12% fat) , Low Fat Refined (35% sugar, 10% fat) diet, High Fat diet (45% fat, as lard and soybean oil) and CAF -- a wide assortment of junk foods from sugary breakfast cereals, to Doritos, to pork rinds, to wedding cake.   If it's carbs and insulin, why don't the rats on the SC get fat?  If it's just refined carbs, why don't the sugar rats get fat?  If it's just the fats, why don't the lardos get fat?   These two groups of rats did gain more weight, but -- as Stephan mentions -- they don't gain a lot more, and more importantly, they don't overeat energy.  The CAF rats, meanwhile, consumed over 30% more food/calories and got a whole lot fatter. 

Gary's Response:  We're looking at this wrong.  Calories are the wrong way to look at this.  











{Original publish date:  2/25/11}


(Hat tip to Beth for bringing this to my attention)

This study used male Wistar rats

{eek ... I'm having flashbacks to a former career!}

This rat is not a genetic mutant designed to be predisposed towards obesity.  It is, however, often used in diet induced obesity (DIO) studies.

Sunday, March 31, 2019

Guyenet v. Taubes ~ Topic 1: "Just 10-20 Calories a Day"



Unless you've been living under a "rock" in the world of nutrition, you're aware that Stephan Guyenet "debated" Gary Taubes on the Joe Rogan Experience podcast.





It took me several days broken up to watch/listen to the whole thing. In a nutshell, it was Science & Substance (Stephan Guyenet) versus Stories & Slander (Gary Taubes).

Rather than summarize, there were several points made that I'd like to address. In no particular order. I'll include this intro with links to prior topics as this series progresses.



Tuesday, December 18, 2018

The $12M NuSI/Ludwig Study ~ Part V: Intake, REE and TEE Measures

Summary:


Continuing on with discussion of:  Effects of a low carbohydrate diet on energy expenditure during weight loss maintenance: randomized trial

Previous posts in this series:
Part I: Critique of the Study Design
Part II: $12 Million for 12% Weight Loss?
Part III: Some "Early" Lessons
Part IV: Insulin Resistance Does Not Hamper Weight Loss

This post should perhaps have come first, but it has taken a while to look deeply at the data for the primary outcome -- total energy expenditure measured by doubly labeled water -- and related outcomes of intake and resting energy expenditure.

In this study, all participants were paid to participate, AND provided free food for ~8 months.  Said food was professionally and meticulously prepped, weighed, measured, individualized to provide each subject with some pretty exact caloric level and macronutrient composition.  The test phase (in other words, the study proper) involved maintaining a consistent weight (to within ±2 kg), for a considerable length of time:  20 weeks, assessed at the midpoint and the end.

In the previous study, a roughly 300 calorie/day increase in TEE was observed during 4 weeks of isocaloric feeding, producing no weight loss.  This was essentially dismissed as "not enough time to show weight loss"  {paraphrase}   ....  So in the current study, the average change in TEE masks the often wild swings of up to 2000 calories per day between 10 week maintenance time points.  If this increase (or decrease) in expenditure is truly real, the subjects would HAVE to have adjusted intake accordingly over the course of several weeks in order to maintain consistent weight.  

This post is about the massive discrepancies between intake and TEE data (also compared with REE), and the inexcusable near-dismissal of these discrepancies in the journal article.


Bottom Line:


Either the DLW-TEE data is (for reasons that can be discussed) unreliable/inaccurate, or the intake data is unreliable. If the TEE data is problematic, how can a result of a difference of means of 250 cal/day be justifiably defended as significant/meaningful, when we see no such difference in intake? If the intake data is problematic, then how can we conclude anything from a "diet comparison", because significant deviations in caloric intake would render the strict macro compositions totally irrelevant. 

Either way, the researchers failed to address this glaring problem with the results study.  Further, the discrepancies in these two data sets are so egregious that it is impossible that both can be considered accurate and reliable.  It has to be one or the other.  Based on the data for Resting Energy Expenditure (REE), the needle points to the intake data as the more reliable measure.



Sunday, December 9, 2018

The $12M NuSI/Ludwig Study ~ Part III: Some "Early" Lessons


SUMMARY




Continuing on with discussion of:
Effects of a low carbohydrate diet on energy expenditure during weight loss maintenance: randomized trial

In Part I, I discussed some issues with methodology, mostly focusing on the reduced Run-In Phase that likely compromised the outcomes irreparably.

In Part II, I highlighted a serious issue with the Run-In Phase, the purpose of which was to produce a somewhat homogeneous "reduced weight state" to test various diets in maintenance of that state.

Ultimately, since randomization to the various test diets occurred after weight loss (PWL) randomization to maintenance test diet would not influence the impact of various BSL (pre-weight loss baseline) measures on the Run-In outcome -- target = 12% ± 2% weight loss -- on a standard composition diet for all:  45% Carb / 30% Fat / 25% Protein.

The researchers appear to have made minimal adjustments, if any, during the Run-In Weight Loss so as to produce a more uniform result.  Rather, the result was a wide range of weight loss (5.6 to 16.0%, roughly 10.5% ± 5%) . 

Thus we have an "accidental" test-within-a-test of the CIH/TWICHOO from these "early" results.

In the end, I offer these scatter plots for all 105 subjects who successfully completed the study, for whom complete data for insulin measures and energy expenditure were available at all time points.


The Carb-Insulin Hypothesis (aka TWICHOO) predicts that weight loss will vary inversely with insulin levels:  The higher the insulin levels, the lesser the weight loss.  The Run-In Phase data supports no such relationship (indeed, if anything, absolute weight loss was greater for those with higher baseline insulin measures.

Meanwhile, differences in weight loss are easily explained by variation in caloric deficit during the calorie restricted Run-In due to coarse estimation of baseline energy expenditure (vs. rigorous measure).

This post expands on some relationships of baseline (BSL) and post-weight loss (PWL) measures as observed during the weight loss portion of the Run-In Phase.

Bottom Line:  Baseline insulin status seems to be irrelevant to weight loss on a "high carb" calorie-restricted diet.  





Monday, December 3, 2018

The $12M NuSI/ Ludwig Study ~ Part II: $12 Million for 12% Weight Loss?

UPDATE:   12/17/2018     Original Posting: 12/3/2018   


During the writing of a new installment in this series, I revisited the following paper:

A randomized study of dietary composition during weight-loss maintenance: Rationale, study design, intervention, and assessment
Yes, folks, so full of themselves were these researchers, that they felt the need to write an entire paper (submitted approx. one year in advance of the "real study") outlining their amazing study. Regarding this post, focusing on the OUTRIGHT FAILURE TO IMPLEMENT THEIR STUDY DESIGN AS STATED, I stress the following from the previous paper.

ONCE AGAIN, the stated goal of the weight loss run in is 12% ... not 10 or 5 or 15 or whatever ... 12%.  I'm just going to quote the relevant part here (cleaned of references, etc. and formatted for readability, all emphasis mine)
Energy intake was restricted to 60% of estimated needs to achieve a target weight loss equating to 12 ± 2% of baseline body weight, with a minimum of 1200 kcal/d. We asked participants to weigh themselves daily using Wi-Fi scales, and the amount of food provided was increased or decreased as necessary to reach the target weight loss over 9 to 10 weeks. 
Previous studies have indicated metabolic adaptation characterized by declines in resting energy expenditure (REE) and TEE with weight loss corresponding to 10% of baseline weight. With 20% weight loss, REE may not decline further, but TEE may continue to decline at a slower rate due to shifts in non-REE. 
We selected the 12 ± 2% weight loss target to ensure significant metabolic adaptation without imposing greater burden (on participants and the study) of a larger weight loss target.
Only they must not have done this as weight loss in this study was roughly 10.5% ± 5%.  FOR SHAME.

/UPDATE


SUMMARY


This post focuses on a critical issue with the Run-In phase of the recently released $12 million dollar NuSI funded study led by David S. Ludwig MD, PhD: Effects of a low carbohydrate diet on energy expenditure during weight loss maintenance: randomized trial. On occasion I reference the 2012 predecessor: Effects of Dietary Composition on Energy Expenditure During Weight-Loss Maintenance.

Virtually every stipulation leading up to the study, the Abstract and Full Text of the journal article, and continuing through to Ludwig's November 28, 2018 response to Kevin Hall in BMJ,  has stated that the MAIN purpose of the Run-In phase was to achieve 12%  ± 2% weight loss.  The study was intended to test the so-called Carb-Insulin Hypothesis during 20 weeks of weight-stable maintenance on diets of varying carbohydrate content.


OUTCOME:
With only 10 weeks of 60% calorie restriction during Run-In, the subjects who completed this phase averaged 10.5% weight loss (std.dev. 1.7%), ranging from only 5.6% up to 16.7% weight loss.  This doesn't change much for analyses of just those completers for whom complete energy data are available (same mean, SD = 1.5%, range 5.9% to 16.0%).

This indicates an unacceptable variation in the post-weight loss (PWL) "biological state", and to use this as the baseline "anchor" for diet comparisons is negated by either Study Design and/or execution thereof.





Wednesday, November 28, 2018

The $12M NuSI/Ludwig Study: Part I: Critique of the Study Design


SUMMARY


The results of the $12 Million Dollar NuSI-sponsored study, headed up by Dr. David S. Ludwig, are finally out.  
Effects of a low carbohydrate diet on energy expenditure during weight loss maintenance: randomized trial
While the good doctor is making the rounds touting them as evidence in support of the Carb-Insulin Hypothesis (TWICHOO in these parts), a review of the raw data made available to the public casts grave doubts on his victory lap.  This study built upon the "promising" results of the 2012 study:  
Effects of Dietary Composition on Energy Expenditure During Weight-Loss Maintenance  

This post focuses on comparisons of Study Design between these two studies, some improvements, and the ultimate failure that renders the primary outcome data suspect, if not outright useless.  (And I don't say that lightly!)


Improvements:


  • Study size
  • Length of time on one test diet (20 weeks) vs. 4 weeks crossover on each diet w/o washout
  • Protein held constant between test diets
  • Macro extremes the same -- e.g. LCHF = 20% carb/60% fat , HCLF = 60% carb / 20% fat
  • Post weight loss (PWL) assessment of TEE
  • Intake adjusted to maintain weight

Detrimental Changes:

  • Drastically altered run-in phase 
    • Shorter in total:  16 weeks vs. 20 weeks
    • No monitoring period before gathering baseline data.
    • No baseline intake assessment
    • Shorter weight loss phase:  10 weeks vs. 12 weeks
    • More varied weight loss and no minimum loss established to be included in the test phase.  In previous study, all subjects had to achieve at least 10% loss, averaged 13.5%.  Current study, losses ranged from 5.6% to 16.0%
    • Only 2 weeks for weight stabilization vs. 4 weeks.
  • Added ad libitum snacks for those who needed to increase caloric intake in maintenance but could not tolerate the larger meals necessary.
  • Assessing post weight loss DLW-TEE during the same two weeks immediately following weight loss that are designated for stabilizing reduced weight.
  • Changing protocol to anchor TEE changes to this flawed PWL measure of TEE instead of baseline per original protocol.

Monday, November 12, 2018

Why Aren't We Taking Anti-Obesity Drugs?



This post was prompted by the following article on Medscape


It is written by Caroline M. Apovian, MD 

I'd encourage you to read this whole thing first, as I'm genuinely interested in that response. Additionally I'm curious as to whether or not your response changes after reading this blog post (or other sources I'm about to link to).